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Curative Embolization of Cerebral AVMs - Glue is Still Relevant

Last update on May 23, 2023

Discover the cases of three patients with cerebral arteriovenous malformations (AVMs) in different locations who underwent curative embolization using glue (NBCA) as the exclusive or main embolic agent. 

Case presentation

Curative Embolization of Cerebral AVMs by Glue

We present three example cases of cerebral AVMs in different locations in which glue (NBCA) was the exclusive (2 cases) or main ( 1 case) embolic agent used. We were able to achieve complete obliteration of AVMs in all the 3 cases.

Intervention

Case 1

Patient presentation

  • Girl, 17-year-old. presented with acute onset headache f/b LOC in Dec, 2021
  • CT/MRI-left posterior pericallosal bleed with IVH
  • DSA → posterior pericallosal & choroidal AVM

 

pericallosal-bleed

 

choroidal-AVM

 

Embolization Session -1 July 2022

  • Pedicle-1: Left Pericallosal

 

pedicle01

 

  • Pedicle-2: peri splenial br.

 

pedicle02

 

  • Pedicle-3: Post. choroidal br.

 

pedicle03

 

  • Pedicle-4: Post. choroidal br.

 

pedicle04

 

Embolization Session -2: March 2023

  • Pedicle-1:Post choroidal-Microcatheter navigation

 

embolization-session2

 

  • Pedicle-2: Pericallosal

 

pedicle2-pericallosal

 

Final control angio

 

Case 2

Patient presentation

  • A 9 yrs boy with sudden severe headache

 

case2

 

case2-02

 

Superselective navigation, glue injection & control angio

superselective-navigation

 

Post embo CT

 

 

post-embo-CT

 

Follow-up MRI @1mo

 

 

follow-up-MRI

 

Case 3

Patient presentation

  • A 32/M; h/o seizures since 2 years
  • Headache f/b tonic posturing f/b LOC.
  • No deficits

 

case3

 

Embolization

  • Pedicle-1: Onyx

 

embo-pedicle-onyx

 

  • Pedicle-2: Glue

 

pedicle2-glue

 

  • Pedicle-3: Glue

 

pedicle3-glue

 

Final Control Angiogram

 

 

final-control-angio
Conclusion
  • Non-adhesive liquid embolic agents have, no doubt, enabled us to achieve curative embolization in a wide range of cerebral AVMs.
  • Yet NBCA (glue) still has its role as illustrated by our cases.
  • Glue can be the primary agent in a certain group of AVMs- e.g choroidal
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